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Researchers may evaluate: Endothelial-cell responses VEGF-associated signaling Cell migration Tube-formation assays Gene-expression changes Receptor-associated pathways These models are useful for studying vascular signaling under controlled conditions and should not be interpreted as evidence of a therapeutic vascular effect

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Number of Vials:4 Vials (Non-Differentiated Results)

Downregulation of CAV1 reduces the levels of GPX4 and FTH1, increases ROS and Fe 2+ concentrations, promotes ferroptosis, and reduces cancer cell migration and invasion, 171 suggesting that the regulatory pathways of CAV1 and ferroptosis may be potential targets for diagnosis and combination therapy strategies in patients with HNSCC