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Several studies, mostly in animals, revealed that inhibitors of DNA methylation, activators and inhibitors of histone modification and modulators of miRNAs reverse a number of pathological changes in the pain epigenome, which are associated with altered expression of pain-relevant genes
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However, unlike HAMLET and alpha1-O, which primarily accumulates in tumor cell nuclei [34], NheA-O remains bound to the cell membrane, making it the primary site of action
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