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Keywords: Ionizing radiation, Reactive oxygen/nitrogen species, Oxidative metabolism, Mitochondria, Genomic instability, adaptive responses, bystander effects 1
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e.g., vorinostat, romidepsin), and bromodomain and extraterminal domain (BET) inhibitors (e.g., JQ1), can reverse the silencing of tumor suppressor genes, suppress oncogenic transcriptional programs, and resensitize tumors to chemotherapy, targeted agents, or immunotherapy