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Similarly, under oxidative stress, ROS can inhibit PTEN, a negative regulator of the PI3K pathway, leading to the activation of the PI3K-AKT signaling axis (196, 197)
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This has been supported by both in vivo studies and patient-based models of different tumor types, including neuroblastoma [26], lung adenocarcinoma [35], and hepatocellular carcinoma [44], which have shown a clear association between hPrdx6 overexpression and increased tumor growth, in addition to poor survival outcomes
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