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Under normal physiological conditions, the glycolytic activity of TECs remains low, and ATP generated through aerobic glycolysis accounts for only a minor proportion (∼4%) of total ATP production ( During AKI, mitochondrial FAO and the transport of FAs into mitochondria are significantly impaired due to mitochondrial dysfunction and disruption of quality control mechanisms ( During both early and late reperfusion phases following IR injury, there is a notable increase in glucose uptake and glycolytic activity in the renal cortex
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Over time, they are susceptible to Ostwald ripening, aggregation, and sedimentation, especially under stress conditions like temperature fluctuation or mechanical vibration [22,66]
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