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Outcome and follow-up The patient was discharged with a blood pressure of 132/66 mmHg on amlodipine, a potassium level of 2.9, and a phosphate level of 1.16

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Figure 1 Hyperglycemia-induced damage In the retina, neurons, glial cells, and vascular cells form an anatomically and functionally interdependent neurovascular unit (NVU), which is essential for maintaining tissue homeostasis and visual function ( Oxidative stress Hyperglycemia induces pronounced retinal oxidative stress, leading to excessive accumulation of reactive oxygen species (ROS) that damage lipids, proteins, and DNA, thereby impairing cellular function and promoting cell death, key events in the onset and progression of DR ( Inflammation Chronic hyperglycemia is a central driver of inflammation in DR, where sustained high glucose induces oxidative stress and accelerates the formation of advanced glycation end-products (AGEs), thereby initiating and perpetuating inflammatory cascades ( Vascular permeability and neovascularization Vascular permeability and neovascularization are central pathological processes driving the onset and progression of DR

including lysis buffers