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specifically, GABA inhibition reverses the pathological pain state in mice, and GABA-A reduces the production of pro-inflammatory mediators, thereby ameliorating the symptoms of inflammation ( 2+ entry, resulting in activation of nuclear factor κB signal, and finally promotes macrophage invasion by inducing CXCL5 and CCL20 expression ( P38/MAPK may be a key player in the link between the GABA/GABA-A axis and inflammation in RA

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[DOI] [PubMed] [Google Scholar] 68.Chacón-Martínez, C