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Under oxidative stress, Nrf2 dissociates from kelch-like ECH-associated protein-1 (Keap1), translocates to the nucleus, and the activated Nrf2 induces the expression of various endogenous antioxidant enzymes [10]
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Understanding how the influenza virus hijacks these lipid metabolic processes is essential for uncovering its pathogenic strategies and identifying novel therapeutic targets
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