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FIGURE 8 Atherosclerosis, a chronic inflammatory disease, may be an ideal environment for the high viral replication capabilities of SARS-CoV-2 in human cells, enhancing hyper-inflammation secondary to immune system dysregulation (Figure 9) that leads to adverse outcomes, as shown in patients with cardiovascular risk factors (258, 259)
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In this mesolimbic pathway, stimulant use can cause changes in the morphology of neurons, regulate dopamine receptor expression, and alter the activity of second messengers, tyrosine hydroxylase, and the transmission of non-dopaminergic neurons [10]
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