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This pathway begins with the conversion of homocysteine to cystathionine

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found that CXCL16-CXCR6 is a critical signal mediating CD8 + T cell migration to the skin in the oxidative stress microenvironment of vitiligo, and they elucidated the signal transduction mechanism by which ER stress induces CXCL16 expression, proposing a new strategy for treating vitiligo by targeting inhibition of ER oxidative stress-CXCL16-CXCR6 (Chen et al., 2019b)